Insulin GMO Creates Type I in Type II Diabetes Simultaneously

"A groundbreaking new study finds synthetic (GMO) insulin is capable of rapidly producing type 1 diabetes in type 2 diabetics."

Here are some clues deep within our DNA/RNA/ATP/ADP that show why this happens...

The repressor protein is produced by a regulator gene. The region of DNA where the repressor protein binds is the operator site. The promoter site is a region of DNA where RNA polymerase can bind. The entire unit (promoter, operator, and genes) is an operon.
The operator acts like a switch that can turn several genes on or off at the same time.
The lac operon is an inducible operon that uses negative control. It is inducible because the structural genes are normally inactive but the presence of lactose induces them to become active. It is negative control because an active repressor prevents transcription.

ATP (Adenosine Triphosphate) 

You can learn all about ATP/ADP here on my blog. http://fightforyourhealth.blogspot.com/2014/02/pesticides-destroy-by-chemically.html

The energy in one glucose molecule is used to produce 36 ATP.  ATP has approximately the right amount of energy for most cellular reactions.
ATP is produced and used continuously. The entire amount of ATP in an organism is recycled once per minute. Most cells maintain only a few seconds supply of ATP.

Transcription of the structural genes of the lac operon also requires reduced levels of glucose. The role of glucose in promoting transcription is discussed later (see Positive and Negative Control of the Lac Operon below).

Positive and Negative Control of the Lac Operon

The lac operon discussed earlier as an example of negative control is also an example of positive control. The cell normally uses glucose as a carbon source. When the level of glucose declines, the level of a signaling molecule called cyclic AMP (cAMP)  increases. Cyclic AMP binds to a protein called CAP (catabolic activator protein) which then binds to the promoter region, causing transcription to occur. Reduced glucose, therefore, promotes the transcription of the lac Z, Y, and A genes. However, because the lac operon is inducible, lactose must be present.
By having both positive and negative control operating at the same time, the structural genes in the lac operon are not active unless the level of glucose is reduced and lactose is available.

Protein Activation

Some proteins are not active when they are first formed. They must undergo modification such as folding, enzymatic cleavage, or bond formation.

Example: Proinsulin is a precursor to the hormone insulin. It must be cleaved into 2 polypeptide chains and then some amino acids must be removed to form insulin.
Many proteins are activated by adding phosphate groups. They can be inactivated by removing phosphate groups. For example, kinases activate by adding phosphate groups and phosphodiesterase inactivates by removing the phosphate groups.

Feedback Control

Some enzymes in a metabolic pathway may be negatively inhibited by products of the pathway.

  In oxidation-reduction reactions, coenzymes often remove electrons from the substrate and pass them to other molecules. Often the electron is added to a proton to form a hydrogen atom before it is passed. In this way, coenzymes serve to carry energy in the form of electrons (or hydrogen atoms) from one compound to another.

Vitamins are small organic molecules required in trace amounts. They usually act as coenzymes or precursors to coenzymes.

http://faculty.clintoncc.suny.edu/faculty/michael.gregory/files/bio%20101/Bio%20101%20Lectures/Gene%20Regulation/gene.htm

"After recombinant (GMO) insulin administration their blood glucose control deteriorated, and their own insulin producing beta cells – as measured by declining C-peptide levels (a marker for the production of natural insulin) – decreased insulin production to a deficiency levels commonly found in type 1 diabetes patients. The average time it took for the patients to develop full blown type 1 diabetes was 7.7 months, with one patient developing the condition within 1.1 months....

Last year, we reported on the dangers of insulin therapy for type 2 diabetics, following the publication of a study comprised of almost 85,000 type 2 diabetic patients that found insulin monotherapy doubled their risk of all-cause mortality, in addition to significantly increasing their risk for diabetes-related complications and cancer. Insulin monotherapy resulted in:

• 2.0 times more myocardial infarctions.
• 1.7 time more major adverse cardiac events
• 1.4 time more strokes
• 3.5 times more renal complications
• 2.1 time more neuropathy
• 1.2 times more eye complications
• 1.4 times more cancer
• 2.2 times more deaths

Now, a new study published in the Journal of Clinical Endocrinology & Metabolism titled, "Insulin administration may trigger type 1 diabetes in Japanese type 2 diabetes patients with type 1 diabetes high-risk HLA class II and the insulin gene VNTR genotype," is shedding light on a possible explanation for why insulin treatment may accelerate morbidity and mortality in type 2 diabetics. The study revealed that giving genetically susceptible type 2 diabetes patients recombinant insulin can trigger their bodies to target their own insulin producing cells for autoimmune destruction, effectively producing 'double diabetes': type 1 and type 2, as a result."

http://www.greenmedinfo.com/blog/gmo-insulin-causes-type-1-diabetes-type-2-diabetics-study-finds