When my fingers were turning black, and I almost lost my arms and legs too...The doctor...Knowing by me telling him, "I was poisoned by pesticide!", many, many times...throughout every doctor visit...and him still saying I have frost bite./..BUT LIVE in Hawai'i?... made more sense to the doctor....
Necrosis is what it was doing and my blood getting clumpy because of that...To not even recognize the entire system of blood in my body was messed up and treat that...He choose to only treat my hands and feet...! He was the one who made me loose my finger tips and joints.
Thank God I changed doctors because was giving me Morphin for pain, and that was slowing down my heart...Just what I needed ...clotted blood and no pushing of it around from the Morphin...
I wanted Percocets...For the pumping...And had to beg...Ended up drinking lots of coffee and Cokes to achieve a strong heart beat...Even then the emergency room could still not find my pulse...They gave me those Perks, and my new doctor was told if I didn't get them I was going to be at the emergency room a loT!...
Couldn't they feel my pain?
I was also dealing with every nerve in my body having it's sheath burned/ripped off...Imagine being skinned alive...but on the insides...
Throw in all the dead tissue, and organs not getting oxygen or a good blood supply...The same thing happening in my fingers and toes was happening to my organs...eg.liver, and kidneys. My joints, every single one, was swollen 2xs it size in some cases...Could not even barely bend...
That is why I say, and did lay in bed and not move for a year...The 1st 3 months I couldn't even lay, or lean back...because it would cut off the blood supply in my arms...Every vein in my body collapsed...and I had no pulse either... :P
So, let's see...
1)
We all (MOST) have morgellons already but do not know it, because it hasn't taken over their bodies (YET)...
2)
85% (MOST) have Toxoplasmosis Ghondi but, do not know it...Also called the Zombie Apocalypse.
3)
ummm...give me a sec...oh...All people are being brainwashed with lies to not know 1 and 2...Which causes ignorance to the reinfection on a daily basis through GM food, and other GM and nanotech items we use daily...Compounded by other chemicals in our environment that weakens our immune systems...including vaccinations and incidental radiation (EMFs, and radioactivity unseen, or smelled around us).....and other GM organisms being created and spread all over the world...(They sell it as organic compost!!!! OMG) that will horizontal combine to form new unknown pathogens to boot!
And do not forget all the other parasites we live with too...Think about the ones dogs and cats get...You and I also have them in us...Ever hear a doctor even mention single celled parasites ever? Not in their vocabulary, or approach to health...
I actually know 2 people with Morgellons, diagnosed by doctors...And another I see has it going on and doesn't know...
They say only 12,000 have it world wide...NOT TRUE...
The same denial is going on with this horribly painful, and life impacting disease cased by genetic modification...as pesticide poisoning...Even going so far as most doctors prescribing ant-psychotic drugs to treat...Saying the patient is insane, and doing all this to themselves, and refusing to look under a microscope and see the fibers-that are communicable to others...just by them falling off their body...If one person in a household gets this...the entire household will get Morgellons too.
Fabric Maui Bus seats...They have all been switched to plastic now...And I do know that the
Dept of Health here has gone to my one friends house to investigate Morgellons with her...
She was had been homeless and sleeping behind a dumpster, and also stayed at the Salvation Army at night too...
So new bus seat covers, and they closed the Salvation Army for sleeping over some lame excuses...hmmm...?
NO WONDER HOMELESS ARE BEING HERDED OUT OF PUBLIC AREAS...To stop the spread????
Homeless due to illnesses is what I've found to be happening...Happened to me in 2001...Homeless due to pesticide poisoning!
part3
part4
" Extensive genetic manipulation of Agrobacterium does have the potential to transform it into an aggressive human pathogen. Genetic engineering is nothing if not enhanced and facilitated horizontal gene transfer and recombination, which is widely acknowledged to be the main route for creating new pathogens. Mae-Wan Ho was among an international panel of scientists have raised this very issue in 1998, calling for a public enquiry into the possible contributions of genetic engineering biotechnology to the aetiology of infectious diseases which has greatly increased since genetic engineering began in the 1970s [16].
The epidemiological data of Morgellons Disease are very incomplete, and the Morgellons Research Foundation’s registry of more than 12 000 families afflicted worldwide is almost certainly only a fraction of the emerging epidemic. Still, it is significant that the majority of the cases are in the United States, the first country to release GM crops and remaining the top producer ever since.
There are other findings implicating Agrobacterium in transgenic plants released into the environment, particularly during the early years of field trials, when knowledge was poor and safety measures not as stringent as they may be today.
Agrobacterium persists in transgenic plants and is a vehicle for gene escape
By the late 1990s, the Agrobacterium vector system became very widely used, and many GM crops created were commercially released.
Scientists at the Kinsealy Research and Development Centre in Dublin, Ireland, and the Scottish Crop Research Institute in Dundee, Scotland, were concerned that the inserted genes in plants would spread to wild populations by cross-pollination or by horizontal gene transfer to unrelated species, which was by then well-documented in the scientific literature.
They considered it “imperative” to address the risk posed in using Agrobacterium as a tool in genetic engineering [17], given its ability to transfer genes to plants. The transformation procedure involves inoculating the cells or tissue explants with Agrobacterium and co-cultivation the plant cells and bacterium for a short period, followed by the elimination of the bacterium with antibiotics.
However, if all the bacteria were not eliminated, then “release of these plants may also result in release of the Agrobacterium [with the foreign genes]”, which will serve as a vehicle for further gene escape, at least to other Agrobacterium strains naturally present in the soil.
Although various antibiotics have been used to eliminate Agrobacterium following transformation, the researchers stated that “very few authors actually test to ensure that the antibiotics succeed.”
The difficulty is compounded because the bacterium can remain latent within the plant tissue. So putting transgenic plant material into culture medium without antibiotics and finding no Agrobacterium is no guarantee that the transgenic plant is free of the bacterium, as was often assumed.
In their study, they investigated the ability of antibiotics to eliminate Agrobacterium tumefaciens after transformation in three model systems: Brassica (mustard), Solanum (potato), and Rubus (raspberry). The antibiotics carbenicillin, cefataxime and ticaracillin were used respectively to eliminate the bacterium at four times the minimum bactericidal concentration, as recommended. They found that none of the antibiotic succeeded in eliminating Agrobacterium.
The contamination levels increased from 12 to 16 weeks to such an extent that transgenic Solanum cultures senesced and died. Contamination in shoot material decreased over 16 to 24 weeks possibly because only the apical node was used in further culture, but even that did not eliminate Agrobacterium from all the samples; 24 percent remained contaminated at 24 weeks.
And the study they talk about here turned out to be another manipulated piece of work... BOGUS! BS...
Another nice company...
On 17th December 2007 Monsanto was found guilty of contempt of the South African Advertising Authority (ASA) for publishing false claims about the safety of GM foods.
In January,2007, Monsanto was fined 15,000 euros (US$19,000 ) in a French court for misleading the public about the environmental impact of herbicide Roundup.
A former chairman of Monsanto Agriculture France was found guilty of false advertising for presenting Roundup as biodegradable and claiming that it left the soil clean after use. Monsanto’s French distributor Scotts France was also fined 15,000 euros.
In 2005 Monsanto was caught smuggling South African produced GM Bollgard cotton seed into Indonesia disguised as rice. Monsanto was fined for bribing Indonesian officials.
In 2006 Monsanto suppressed evidence of serious damage to the liver and kidneys of rats in their MON 863 GM maize trials until ordered to release this evidence by a German Court.
In June, 2007, a second peer-reviewed case involving another variation of Monsanto’s GM maize, namely, NK 603, has been shown by studies to be potentially toxic to humans. NK 603 has been approved for food, feed, processing, and propagation in Europe and the Philippines The new research, carried out by the French scientific research institute CRIGEN, involves biotech firm Monsanto’s NK 603 GMO corn (marketed commercially under the name Round-up Ready).
Rats that were fed GM maize showed significant differences in measurements, as well as significant weight differences compared to those fed with normal maize. Almost 70 statistically significant differences were observed and reported - 12 for hematology parameters, 18 for clinical chemistry parameters, nine for urine chemistry parameters, six for the organ weights (brain, heart, liver), 14 for body weights and body weight changes, and eight for food consumption. toxicity, The most alarming was the diminished brain size. Scientists warned that diminished brain size sent out a urgent danger warning for growing children fed `GM food.
Again in 2008 many trials against Monsanto, accusing them and Bayer too, to be also responsable for the die off of honey-bees. And so on...
German government has in 2009 forbidden by law the cultivation of Monsanto maize (Mon 810) in Germany!!
At least the German government (before elections) is much wiser then US government with their insane Monsanto
lobby. Now also other European countries will follow after Germany has started to ban Monsanto...And USA
http://www.worldhealthnet.tv/video/jeff-smith-the-effects-of-genetically-modified-foods
US Doctor`s association calls for Moratorium on GMO Foods by F. William Engdahl
In a just-released position paper on GMO foods, the American Academy of Environmental Medicine (AAEM) states that ‘GM foods pose a serious health risk’ and calls for a moratorium on GMO foods. Citing several animal studies, the AAEM concludes ‘there is more than a casual association between GMO foods and adverse health effects’ and that ‘GM foods pose a serious health risk in the areas of toxicology, allergy and immune function, reproductive health, and metabolic, physiologic and genetic health.’ The report is a devastating blow to the multibillion dollar international agribusiness industry, most especially to Monsanto Corporation, the world’s leading purveyor of GMO seeds and related herbicides.
http://www.morgellons-research.org/morgellons/one-of-the-worst-morgellons-cases.htm
Agrobacterium is released in nature with a transgenic plasmid is capable of passing varied traits to other bacteria so the actual pathogen that causes Morgellons is yet to be classified but it is safe to say it is a Rhizobiales order bacteria, you can still treat it. It should also be noted that bacteria in nature are in constant evolution so subsequent type pathogens of various strains are possible. In Morgellons there is fiber growth in the tissues of infected people. The fibers have been determined to be keratanized cells by Charles Holman Foundation researchers, your fingernails and toenails are keratinized cells. There is also a collagen content in the fibers.
When any cells grow in our bodies it is because the information found in the genetic material, DNA, has defined the instruction for exactly what is created. When fibers grow out of someone then the conclusion should be that the genome has been altered and a gene has been inserted from another organism . The MD producing gene is using building blocks found in our bodies. Agrobacteriium is a naturally occurring bacteria and is altered to become a tool for genetic engineering. Agrobacterium causing Crown Gall disease in plants shows creating a tumor tissue in the plant in a gall shape that sequester bacterial concentrations. This similarly created genetic callus material is what we see in Morgellons presentations as the hard debris that come from lesions and out of other areas on the skin. The fibers grow because of a transgenic structure using our bodies building materials. They usually grow out of areas where the genetically produced tumor tissue material has infected.
The genetic engineering process using Agrobacterium with T-DNA has yielded a bacteria that in itself, or by gene transfer into another bacteria such as Bartonella, so that bacteria now becomes the MD pathogen that causes MD. The specific gene that causes the fiber growth is not yet known perhaps from a fungus. There is also the possibility that Bartonella receives a gene from another infective pathogen inside the body and then uses it's ability to transfer that into human cells .
The credible evidence strongly suggests that Morgellons Disease is a manifestation of genetic manipulation from a Rhizobiales order bacteria with an unknown genetic variant. Since Agrobacterium and Bartonella are so closely related the exact pathogen could be a Bartonella like organism. They are Rhizobiales order alpha proteobacteria. They are gram-negative and share some genes. Bartonella is very difficult to remove from the body. Morgellons typically exhibits this quality of being very difficult to remove from the body as well. In my research I looked at pictures of Bartonella lesions and MD lesions and found them to be very similar. In Bartonella you get "red track marks" on the skin, you also get these in MD. It is not only that the MD pathogen is tenacious like Bartonella and has very similar lesions but it is also that is produces the internal growth of tumor tissue similar to the tumor tissue in Crown Gall disease.
http://www.lyme-morgellons.com/morgellons.html
Based on past detailed studies of rod-shaped bacteria such as Escherichia coli and Bacillus subtilis, it has been assumed that most bacteria grow by binary fission, a dispersed mode of growth involving insertion of new cell wall material uniformly along the long axis of the cell. Growth requires breaking the cell wall at numerous places along the cylinder to allow insertion of new cell wall material, enabling uniform elongation of the cell, with the process culminated by cleavage at the mid-point of the cell to create two symmetric new cells.
The new research published today, Jan. 17, in Proceedings of the National Academy of Sciences reports on the surprising discovery that cell growth in a large group of rod-shaped bacteria occurs by insertion of new cell wall material only at a single end, or pole, of the cell rather than by the dispersed mode of growth. The cell wall of the progenitor cell remains largely intact, and all of the new cell wall material is partitioned into the new cell.
Polar growth of four bacterial species -- the plant symbiont Sinorhizobium meliloti, the plant pathogen Agrobacterium tumefaciens and the human pathogens Brucella abortus and Ochrobactrum anthropi -- was observed using time-lapse microscopy and transmission electron microscopy. The four related bacteria used in the study are all members of a large and diverse class of bacteria called the Alphaproteobacteria. The results reported suggest that polar growth is broadly distributed among many different bacterial taxa, including groups outside the Alphaproteobacteria.
There could be a number of reasons why polar growth emerged and has remained conserved and persistent in bacteria, the researchers believe. The process may act as an aid in anchoring damaged material to only the aging mother cell; it could serve as a tool for conservation of energy by constraining growth to a single region of the cell; and ensuring that newborn cells are composed of newly synthesized outer membrane proteins may help pathogens avoid detection by host immune systems.
"As a consequence of polar growth, the two bacterial cells are actually markedly different," said lead author Yves Brun, the Clyde Culbertson Professor of Biology in IU Bloomington's College of Arts and Sciences. "One cell contains all of the old cell wall and surface molecules, including those with damage. In contrast, the other cell is composed of newly synthesized, relatively pristine material."
Ensuring that some cells are composed of newly synthesized surface molecules may help bacteria vary their surface composition, and the ability to do so rapidly is thought to be advantageous for adapting to new environments. Since the defense systems of many plant and animal hosts recognize bacterial cell surfaces, rapid modification of the cell surface may allow bacteria like those used in the experiments to evade detection by the host cell's defense systems.
"These findings make it abundantly clear that the widely accepted binary fission model is not a general rule and suggest that polar growth may be broadly distributed," said IU biology professor Clay Fuqua, one of the IU co-authors. "Therefore, future work aimed at understanding the molecular mechanism underlying polar growth should provide attractive targets for the development of new antibacterial strategies."
Understanding the mechanisms of bacterial growth has enabled advances in strategies to limit the proliferation of bacteria that cause disease. Penicillin, for example, targets actively growing cells by directly inhibiting the proteins responsible for the synthesis of the cell wall and that are required for cell growth. New insights into bacterial cell growth have also been utilized to promote growth of certain bacteria used in oil spill remediation and eradicating disease-carrying mosquitoes.
http://newsinfo.iu.edu/news/page/normal/20892.html
FOR IMMEDIATE RELEASE
Jan. 17, 2012
BLOOMINGTON, Ind. -- An international team of microbiologists led by Indiana University researchers has identified a new bacterial growth process -- one that occurs at a single end or pole of the cell instead of uniform, dispersed growth along the long axis of the cell -- that could have implications in the development of new antibacterial strategies.
"A different but related issue is the inability of plant tissues to respond to culture manipulations for the desired outcome (Benson 2000a). Under normal tissue culture conditions, even without transformation, many plant species fail to respond to culture manipulations (Benson 2000a). This phenomenon, referred to as “in vitro recalcitrance”, has been ascribed to several factors including cellular incompetence and necrosis but, again, the exact reason(s) for this problem remain unclear.
The three common problems described above, (1) browning/necrosis of transformed cells/tissues, (2) shoot escapes in transgenesis, and (3) in vitro recalcitrance, severely limit the number of transgenic plants that can be regenerated. Although seemingly unrelated, recent research is beginning to unravel a common factor underlying these problems, the production of ROS, which can cause growth inhibition, cell death, or alter plant metabolic pathways leading to poor regeneration of plants and the production of shoot escapes."
Biological Functions of Antioxidants in Plant Transformation - Science News - redOrbit
By Dan, Yinghui Abstract Browning and necrosis of transformed cells/ tissues, and difficulty to regenerate transgenic plants from the transformed cells/tissues (recalcitrance) are common in Agrobacterium-mediated transformation process in many plant species.
REDORBIT.COM
http://www.redorbit.com/news/science/1523653/biological_functions_of_antioxidants_in_plant_transformation/#3hfqtXO8he91bP8D.99
Autoimmunity crisis...
Caused by trying to modify Strawberries?
"Decomposition of peroxides by heating or by transition metal ion catalysis can generate both peroxyl and alkoxyl radicals (Halliwell et al. 1995). The sites and actions of antioxidants in living cells. Main antioxidant actions include scavenging ROS or free radicals, inhibiting the generation of ROS, and chelating metals, as well as their effects on cell signaling pathways and on gene expression (Halliwell et al. 1995; Soobratteea et al. 2005). However, it seems that antioxidants that interfere with the activity of OH’ do not by direct OH’ scavenging, but by scavenging or blocking the formation of its precursors (O^sup -^^sub 2^, H^sub 2^O^sub 2^, HOC1, ONOO’) and/or by binding the transition metal ions needed for OH’ formation from O^sup -^^sub 2^ and H^sub 2^O^sub 2^ (Halliwell et al. 1995). In addition, many lipid-soluble chain-breaking antioxidants can have prooxidant properties, which induce oxidative stress either through creating reactive oxygen species or inhibiting antioxidant systems, under certain circumstances in vitro, often because they can bind Fe(III) or Cu(II) ions and reduce them to Fe^sup 2+^Or Cu+ (Mukai et al. 1993).
Antioxidants act as scavengers of ROS, such as the peroxyl radical either in aqueous phase (e.g., with radicals from DNA, thiols, and proteins) or in hydrophobic phase (food lipid, membrane, lipoprotein interior) (Halliwell et al. 1995). For example, glutathione reacts rapidly with free radicals generating from an attack of OH’ on DNA, at rate constants of about 10^sup 7^-10^sup 8^ M^sup -1^ s^sup -1^ (von Sonntag 1987; Fahey 1988). Other antioxidants, e.g., scavengers of the peroxyl radical such as chain breaking antioxidants (propyl gallate and alpha-tocopherol) that are inhibitors of lipid peroxidation, could act hydrophobically in lipids, cell membranes, and interior structure of lipoproteins.
Major Problems and Antioxidants Used in Plant Tissue Culture and Transformation
Hyperhydricity. Hyperhydricity, a physiological disorder occurring in plant tissue cultures, is associated oxidative damage (Chakrabarty et al. 2006). Hyperhydricity results in a general decrease in concentrations of reduced and oxidized pyridine nucleotides, reflecting a reduction in metabolic activity (Chakrabarty et al. 2006). The activities of antioxidant enzymes, such as superoxide dismutase (SOD), catalase, ascorbate peroxidase, and glutathione reductase, were higher in hyperhydric leaves than in normal leaves, indicating that hyperhydricity was associated with oxidative stress (Chakrabarty et al. 2006). Measurements of chlorophyll fluorescence provided evidence of oxidative damage to the photosynthetic machinery in the hyperhydric leaves because the photochemical efficiency of photosystem II, the effective quantum efficiency, and photochemical quenching were all lower in the hyperhydric leaves (Chakrabarty et al. 2006). Toth et al. (2004) reported that the inclusion of glutathione in a culture medium could suppress the hyperhydricity of calli, thereby promoting regeneration of plantlets in a desiccation-tolerant plant, Ramonda myconi.
Recalcitrance. In vitro recalcitrance of plants has been associated with ROS production. Higher levels of free radical activity were found in recalcitrant genotypes of potato and grape as well as in nonembryogenic calli (rice) and reduced embryogenic capacity calli (carrot) compared to responsive genotypes and embryogenic calli, respectively, (Benson et al., 1992; Benson and Roubelakis-Angelakis 1992, 1994; Bailey et al. 1994; Bremner et al. 1997; Deighton et al. 1997). The production of the free radicals might lead to an increase in lipid peroxidation and subsequently have a negative effect on the morphogenetic capacity of plant tissue cultures (Benson 2000b).
Production of H^sub 2^O^sub 2^ coincided with the emergence of meristemoids and the formation of bud primordia in strawberry morphogenic calli (Tian et al. 2003). High O^sup -^^sub 2^ level, low H^sub 2^O^sub 2^ level, and little or no SOD activity were detected in calli possessing low organogenesis capacity. Adding N,N- diethyldithiocarbonate, an SOD inhibitor, to the regeneration medium promoted O^sup -^^sub 2^ production, inhibited H^sub 2^O^sub 2^ production and decreased the regeneration percentage, whereas the exogenous addition of H^sub 2^O^sub 2^ slightly promoted the potential for regeneration of shoot buds."
or tobacco? or GMO yeast?
"There is quite a diversity of strange material that has unearthed itself from my body. Some of it may be what I call "throwbacks" to genetics used in original bioengineered mixtures. For more information read my "Throwback Collection of Morgellons Specimens." Other Morgellons specimens look like they are straight from a bad Sci-Fi movie or the lab of a mad scientist. The sad part of it is that I seem be to correct about the origins of Morgellons. I guess it's true that art imitates life.
Those of us with Morgellons Disease have found ourselves living in an unthinkable and ignored alternate reality. It is quite possible that everyone has Morgellons and those of us with severe reactions to it are the only people who notice. That is something for everyone to ponder. Clifford Carnicom has preliminary research that points to this conclusion. Take a look at my simple "Hairbrush Test" paper and check yourself for the tell tale signs of nanofibers.
http://www.morgellonsexposed.com/pjOddities.htm
Clifford E Carnicom Oct 15, 2011 (edited Dec 1, 2011)
Morgellons: A Thesis
Abstract:
A substantial body of research has accumulated to make the case that the underlying organism (i.e., pathogen) of the so-called "Morgellons" condition, as identified by this researcher, is using the iron from human blood for its own growth and existence. It will also be shown that the bio-chemical state of the blood is being altered in the process. The implications of this thesis are severe as this alteration affects, amongst other things, the ability and capacity of the blood to bind to oxygen. Respiration is the source of energy for the body.
This change is also anticipated to increase the number of free radicals and to increase acidity in the body. This process also requires and consumes energy from the body to take place; this energy supports the growth and proliferation of the organism. The changes in the blood are anticipated to increase its combination with respiratory inhibitors and toxins.The changes under evaluation may occur without any obvious outward symptoms. It is also anticipated that there are consequences upon metabolism and health that extend beyond the functions of the blood. This change represents essentially a systemic attack upon the body, and the difficulties of extinction of the organism are apparent. Physiological conditions that are in probable conjunction with the condition are identified. Strategies that may be beneficial in mitigating the severity of the condition are enumerated.
http://www.carnicominstitute.org/articles/bio2011-6.htm
Morgellons: a novel dermatological perspective as the multisystem infective disease borreliosis [v1; ref status: awaiting peer review,http://f1000r.es/116]
Peter Mayne1, John S English2, Edward J Kilbane3, Jennie M Burke4, Marianne J Middelveen1, Raphael B Stricker1
Abstract
Morgellons disease (MD) is a term that has been used in the last decade to describe filaments that can be found in human epidermis. It is the subject of considerable debate within the medical profession and is often labeled as delusions of parasitosis or dermatitis artefacta. This view is challenged by recent published scientific data put forward between 2011-2013 identifying the filaments found in MD as keratin and collagen based and furthermore associated with spirochetal infection. The novel model of the dermopathy put forward by those authors is further described and, in particular, presented as a dermal manifestation of the multi-system disease complex borreliosis otherwise called Lyme disease. A differential diagnosis is drawn from a dermatological perspective. The requirements for a diagnosis of delusional disorder from a psychiatric perspective are clarified and the psychological or psychiatric co-morbidity that can be found with MD cases is presented. A concurrent case incidence is also included. Management of the multisytem disease complex is discussed both in general and from a dermatological perspective. Finally replacement of the term ‘Morgellons’ by ‘borrelial dermatitis’ is proposed within the profession.
Pictured: Alice firemen suited up in their Haz-Mat gear Tuesday after a woman possibly exposed bank tellers to a contagious disease by rubbing a canister on her scabby arms. Photo by OFELIA GARCIA HUNTER
Haz-Mat team cleans up teller lane after woman threatens staff
Ofelia Garcia Hunter, Alice Echo-News Journal
A woman claiming to have Morgellons, a skin disease, drove through the Wells Fargo motor bank downtown and threatened the tellers by rubbing the bank canister on her sores and sending it through the tube.
"A customer went through the drive-thru and threatened the bank staff to give them a contagious disease," said investigator David Valadez with the police department.
Police said the lady, who was not identified and has not been arrested, had threatened the bank before.
About 2 p.m. Tuesday, the woman threatened the tellers at the motor bank with her disease called Morgellons.Police and the fire department blocked off about four lanes and the Alice Fire Department Haz-Mat team suited up and decontaminated the area, including the bank canister she apparently rubbed on her sores and the lane she used, police said.
According to the Internet, "Morgellons is an unexplained and debilitating condition that has emerged as a public health concern…Persons who suffer from this condition report a range of coetaneous symptoms including crawling, biting and stinging sensations; granules, threads or black speck-like materials on or beneath the skin; and/or skin lesions (e.g., rashes or sores) and some sufferers also report systemic manifestations such as fatigue, mental confusion, short term memory loss, joint pain and changes in vision."
Necrosis is what it was doing and my blood getting clumpy because of that...To not even recognize the entire system of blood in my body was messed up and treat that...He choose to only treat my hands and feet...! He was the one who made me loose my finger tips and joints.
Thank God I changed doctors because was giving me Morphin for pain, and that was slowing down my heart...Just what I needed ...clotted blood and no pushing of it around from the Morphin...
I wanted Percocets...For the pumping...And had to beg...Ended up drinking lots of coffee and Cokes to achieve a strong heart beat...Even then the emergency room could still not find my pulse...They gave me those Perks, and my new doctor was told if I didn't get them I was going to be at the emergency room a loT!...
Couldn't they feel my pain?
I was also dealing with every nerve in my body having it's sheath burned/ripped off...Imagine being skinned alive...but on the insides...
Throw in all the dead tissue, and organs not getting oxygen or a good blood supply...The same thing happening in my fingers and toes was happening to my organs...eg.liver, and kidneys. My joints, every single one, was swollen 2xs it size in some cases...Could not even barely bend...
That is why I say, and did lay in bed and not move for a year...The 1st 3 months I couldn't even lay, or lean back...because it would cut off the blood supply in my arms...Every vein in my body collapsed...and I had no pulse either... :P
So, let's see...
1)
We all (MOST) have morgellons already but do not know it, because it hasn't taken over their bodies (YET)...
2)
85% (MOST) have Toxoplasmosis Ghondi but, do not know it...Also called the Zombie Apocalypse.
3)
ummm...give me a sec...oh...All people are being brainwashed with lies to not know 1 and 2...Which causes ignorance to the reinfection on a daily basis through GM food, and other GM and nanotech items we use daily...Compounded by other chemicals in our environment that weakens our immune systems...including vaccinations and incidental radiation (EMFs, and radioactivity unseen, or smelled around us).....and other GM organisms being created and spread all over the world...(They sell it as organic compost!!!! OMG) that will horizontal combine to form new unknown pathogens to boot!
And do not forget all the other parasites we live with too...Think about the ones dogs and cats get...You and I also have them in us...Ever hear a doctor even mention single celled parasites ever? Not in their vocabulary, or approach to health...
I actually know 2 people with Morgellons, diagnosed by doctors...And another I see has it going on and doesn't know...
They say only 12,000 have it world wide...NOT TRUE...
The same denial is going on with this horribly painful, and life impacting disease cased by genetic modification...as pesticide poisoning...Even going so far as most doctors prescribing ant-psychotic drugs to treat...Saying the patient is insane, and doing all this to themselves, and refusing to look under a microscope and see the fibers-that are communicable to others...just by them falling off their body...If one person in a household gets this...the entire household will get Morgellons too.
Fabric Maui Bus seats...They have all been switched to plastic now...And I do know that the
Dept of Health here has gone to my one friends house to investigate Morgellons with her...
She was had been homeless and sleeping behind a dumpster, and also stayed at the Salvation Army at night too...
So new bus seat covers, and they closed the Salvation Army for sleeping over some lame excuses...hmmm...?
NO WONDER HOMELESS ARE BEING HERDED OUT OF PUBLIC AREAS...To stop the spread????
Homeless due to illnesses is what I've found to be happening...Happened to me in 2001...Homeless due to pesticide poisoning!
From what I have read this morning...
We all have it...
and like I react strongly to pesticides...Some react to DNA modified / RNA interfered differently from others, and this shows GMOs ~ DNA manipulation and RNA interference are the reason for illness here...especially if they have found to be (see below in post this quote)..."In Morgellons there is fiber growth in the tissues of infected people. The fibers have been determined to be keratanized cells by Charles Holman Foundation researchers, your fingernails and toenails are keratinized cells. There is also a collagen content in the fibers.
When any cells grow in our bodies it is because the information found in the genetic material, DNA, has defined the instruction for exactly what is created. When fibers grow out of someone then the conclusion should be that the genome has been altered and a gene has been inserted from another organism ."
I believe that morgellons is a disease of the Fascia...exactly as they have found here which stopped all growth of the stem cells of the Fascia...by playing with Agrobacterium...
ASK SOME QUESTIONS...?
The researchers treated some human stem cells with a small amount of RNA designed to suppress HERV-H. Doing so, they found, removed the stem cell's ability to develop into any human cell—instead they would only grow into cells that resembled fibroblasts—cells normally found in connective tissue. A closer look revealed that suppressing HERV-H also suppressed the production of proteins necessary for pluripotency. Thus, at least in humans, the remnant viral DNA appears to be necessary for normal human development—without it, human life would be impossible.
Because of the role HERV-H plays in pluripotency, its possible other remnant viral DNA plays a role in human development as well,
We all have it...
and like I react strongly to pesticides...Some react to DNA modified / RNA interfered differently from others, and this shows GMOs ~ DNA manipulation and RNA interference are the reason for illness here...especially if they have found to be (see below in post this quote)..."In Morgellons there is fiber growth in the tissues of infected people. The fibers have been determined to be keratanized cells by Charles Holman Foundation researchers, your fingernails and toenails are keratinized cells. There is also a collagen content in the fibers.
When any cells grow in our bodies it is because the information found in the genetic material, DNA, has defined the instruction for exactly what is created. When fibers grow out of someone then the conclusion should be that the genome has been altered and a gene has been inserted from another organism ."
I believe that morgellons is a disease of the Fascia...exactly as they have found here which stopped all growth of the stem cells of the Fascia...by playing with Agrobacterium...
ASK SOME QUESTIONS...?
The researchers treated some human stem cells with a small amount of RNA designed to suppress HERV-H. Doing so, they found, removed the stem cell's ability to develop into any human cell—instead they would only grow into cells that resembled fibroblasts—cells normally found in connective tissue. A closer look revealed that suppressing HERV-H also suppressed the production of proteins necessary for pluripotency. Thus, at least in humans, the remnant viral DNA appears to be necessary for normal human development—without it, human life would be impossible.
Because of the role HERV-H plays in pluripotency, its possible other remnant viral DNA plays a role in human development as well,
Our Fascia is being affected no doubt...Modified already, and just starting to get reported, but has been around more then 15 years on the web...
THIS WILL GET YOUR ATTENTION
This article is taken from ISIS (Institute for Science in Society)...
THIS WILL GET YOUR ATTENTION
This article is taken from ISIS (Institute for Science in Society)...
A very anti-GMO group of scientists...
And here is part one of a video Mae Wan Ho has made about the Agrobacterium and Morgellons...
...
Morgellons and Agrobacterium: The Geneticist who Warned the World all
part1
And here is part one of a video Mae Wan Ho has made about the Agrobacterium and Morgellons...
...
Morgellons and Agrobacterium: The Geneticist who Warned the World all
part1
part2
part3
part4
" Extensive genetic manipulation of Agrobacterium does have the potential to transform it into an aggressive human pathogen. Genetic engineering is nothing if not enhanced and facilitated horizontal gene transfer and recombination, which is widely acknowledged to be the main route for creating new pathogens. Mae-Wan Ho was among an international panel of scientists have raised this very issue in 1998, calling for a public enquiry into the possible contributions of genetic engineering biotechnology to the aetiology of infectious diseases which has greatly increased since genetic engineering began in the 1970s [16].
The epidemiological data of Morgellons Disease are very incomplete, and the Morgellons Research Foundation’s registry of more than 12 000 families afflicted worldwide is almost certainly only a fraction of the emerging epidemic. Still, it is significant that the majority of the cases are in the United States, the first country to release GM crops and remaining the top producer ever since.
There are other findings implicating Agrobacterium in transgenic plants released into the environment, particularly during the early years of field trials, when knowledge was poor and safety measures not as stringent as they may be today.
Agrobacterium persists in transgenic plants and is a vehicle for gene escape
By the late 1990s, the Agrobacterium vector system became very widely used, and many GM crops created were commercially released.
Scientists at the Kinsealy Research and Development Centre in Dublin, Ireland, and the Scottish Crop Research Institute in Dundee, Scotland, were concerned that the inserted genes in plants would spread to wild populations by cross-pollination or by horizontal gene transfer to unrelated species, which was by then well-documented in the scientific literature.
They considered it “imperative” to address the risk posed in using Agrobacterium as a tool in genetic engineering [17], given its ability to transfer genes to plants. The transformation procedure involves inoculating the cells or tissue explants with Agrobacterium and co-cultivation the plant cells and bacterium for a short period, followed by the elimination of the bacterium with antibiotics.
However, if all the bacteria were not eliminated, then “release of these plants may also result in release of the Agrobacterium [with the foreign genes]”, which will serve as a vehicle for further gene escape, at least to other Agrobacterium strains naturally present in the soil.
Although various antibiotics have been used to eliminate Agrobacterium following transformation, the researchers stated that “very few authors actually test to ensure that the antibiotics succeed.”
The difficulty is compounded because the bacterium can remain latent within the plant tissue. So putting transgenic plant material into culture medium without antibiotics and finding no Agrobacterium is no guarantee that the transgenic plant is free of the bacterium, as was often assumed.
In their study, they investigated the ability of antibiotics to eliminate Agrobacterium tumefaciens after transformation in three model systems: Brassica (mustard), Solanum (potato), and Rubus (raspberry). The antibiotics carbenicillin, cefataxime and ticaracillin were used respectively to eliminate the bacterium at four times the minimum bactericidal concentration, as recommended. They found that none of the antibiotic succeeded in eliminating Agrobacterium.
The contamination levels increased from 12 to 16 weeks to such an extent that transgenic Solanum cultures senesced and died. Contamination in shoot material decreased over 16 to 24 weeks possibly because only the apical node was used in further culture, but even that did not eliminate Agrobacterium from all the samples; 24 percent remained contaminated at 24 weeks.
And the study they talk about here turned out to be another manipulated piece of work... BOGUS! BS...
Agrobacterium & Morgellons Disease, A GM Connection?
Preliminary findings suggest a link between Morgellons Disease and Agrobacterium, a soil bacterium extensively manipulated and used in making GM crops; has genetic engineering created a new epidemic?
A fully illustrated and referenced version is posted on ISIS members’ website. Details here.
An electronic version of the complete report, or any other complete ISIS report, can be sent to you via e-mail for a donation of £3.50. Please e-mail the title of the report to: report@i-sis.org.uk
CDC launch investigation on Morgellons’ Disease
The Centers for Disease Control (CDC) in the United States announced the launch of an investigation on ‘Morgellons Disease’ in January 2008 [1], after receiving thousands of complaints from people with this bewildering condition, which it describes as follows [2]: “Persons who suffer from this unexplained skin condition report a range of cutaneous (skin) symptoms including crawling, biting and stinging sensations; granules, threads, fibers, or black speck-like materials on or beneath the skin, and/or skin lesions (e.g., rashes or sores). In addition to skin manifestations, some sufferers also report fatigue, mental confusion, short term memory loss, joint pain, and changes in visions.”
Morgellons Disease first became known in 2001, when Mary Leitao created a web site describing the illness in her young son, which she named after a 17th century medical study in France describing similar symptoms [3]. Until then, people with Morgellons Disease have been diagnosed as cases of “delusional parasitosis”, in which the symptoms are deemed entirely imaginary, and lesions allegedly due to self-inflicted wounds.
Indeed, the debate over Morgellons Disease has continued in the pages of medical and scientific journals right up to the CDC’s announcement [4-7]
On 17th December 2007 Monsanto was found guilty of contempt of the South African Advertising Authority (ASA) for publishing false claims about the safety of GM foods.
In January,2007, Monsanto was fined 15,000 euros (US$19,000 ) in a French court for misleading the public about the environmental impact of herbicide Roundup.
A former chairman of Monsanto Agriculture France was found guilty of false advertising for presenting Roundup as biodegradable and claiming that it left the soil clean after use. Monsanto’s French distributor Scotts France was also fined 15,000 euros.
In 2005 Monsanto was caught smuggling South African produced GM Bollgard cotton seed into Indonesia disguised as rice. Monsanto was fined for bribing Indonesian officials.
In 2006 Monsanto suppressed evidence of serious damage to the liver and kidneys of rats in their MON 863 GM maize trials until ordered to release this evidence by a German Court.
In June, 2007, a second peer-reviewed case involving another variation of Monsanto’s GM maize, namely, NK 603, has been shown by studies to be potentially toxic to humans. NK 603 has been approved for food, feed, processing, and propagation in Europe and the Philippines The new research, carried out by the French scientific research institute CRIGEN, involves biotech firm Monsanto’s NK 603 GMO corn (marketed commercially under the name Round-up Ready).
Rats that were fed GM maize showed significant differences in measurements, as well as significant weight differences compared to those fed with normal maize. Almost 70 statistically significant differences were observed and reported - 12 for hematology parameters, 18 for clinical chemistry parameters, nine for urine chemistry parameters, six for the organ weights (brain, heart, liver), 14 for body weights and body weight changes, and eight for food consumption. toxicity, The most alarming was the diminished brain size. Scientists warned that diminished brain size sent out a urgent danger warning for growing children fed `GM food.
Again in 2008 many trials against Monsanto, accusing them and Bayer too, to be also responsable for the die off of honey-bees. And so on...
German government has in 2009 forbidden by law the cultivation of Monsanto maize (Mon 810) in Germany!!
At least the German government (before elections) is much wiser then US government with their insane Monsanto
lobby. Now also other European countries will follow after Germany has started to ban Monsanto...And USA
http://www.worldhealthnet.tv/video/jeff-smith-the-effects-of-genetically-modified-foods
US Doctor`s association calls for Moratorium on GMO Foods by F. William Engdahl
In a just-released position paper on GMO foods, the American Academy of Environmental Medicine (AAEM) states that ‘GM foods pose a serious health risk’ and calls for a moratorium on GMO foods. Citing several animal studies, the AAEM concludes ‘there is more than a casual association between GMO foods and adverse health effects’ and that ‘GM foods pose a serious health risk in the areas of toxicology, allergy and immune function, reproductive health, and metabolic, physiologic and genetic health.’ The report is a devastating blow to the multibillion dollar international agribusiness industry, most especially to Monsanto Corporation, the world’s leading purveyor of GMO seeds and related herbicides.
http://www.morgellons-research.org/morgellons/one-of-the-worst-morgellons-cases.htm
Agrobacterium is released in nature with a transgenic plasmid is capable of passing varied traits to other bacteria so the actual pathogen that causes Morgellons is yet to be classified but it is safe to say it is a Rhizobiales order bacteria, you can still treat it. It should also be noted that bacteria in nature are in constant evolution so subsequent type pathogens of various strains are possible. In Morgellons there is fiber growth in the tissues of infected people. The fibers have been determined to be keratanized cells by Charles Holman Foundation researchers, your fingernails and toenails are keratinized cells. There is also a collagen content in the fibers.
When any cells grow in our bodies it is because the information found in the genetic material, DNA, has defined the instruction for exactly what is created. When fibers grow out of someone then the conclusion should be that the genome has been altered and a gene has been inserted from another organism . The MD producing gene is using building blocks found in our bodies. Agrobacteriium is a naturally occurring bacteria and is altered to become a tool for genetic engineering. Agrobacterium causing Crown Gall disease in plants shows creating a tumor tissue in the plant in a gall shape that sequester bacterial concentrations. This similarly created genetic callus material is what we see in Morgellons presentations as the hard debris that come from lesions and out of other areas on the skin. The fibers grow because of a transgenic structure using our bodies building materials. They usually grow out of areas where the genetically produced tumor tissue material has infected.
The genetic engineering process using Agrobacterium with T-DNA has yielded a bacteria that in itself, or by gene transfer into another bacteria such as Bartonella, so that bacteria now becomes the MD pathogen that causes MD. The specific gene that causes the fiber growth is not yet known perhaps from a fungus. There is also the possibility that Bartonella receives a gene from another infective pathogen inside the body and then uses it's ability to transfer that into human cells .
The credible evidence strongly suggests that Morgellons Disease is a manifestation of genetic manipulation from a Rhizobiales order bacteria with an unknown genetic variant. Since Agrobacterium and Bartonella are so closely related the exact pathogen could be a Bartonella like organism. They are Rhizobiales order alpha proteobacteria. They are gram-negative and share some genes. Bartonella is very difficult to remove from the body. Morgellons typically exhibits this quality of being very difficult to remove from the body as well. In my research I looked at pictures of Bartonella lesions and MD lesions and found them to be very similar. In Bartonella you get "red track marks" on the skin, you also get these in MD. It is not only that the MD pathogen is tenacious like Bartonella and has very similar lesions but it is also that is produces the internal growth of tumor tissue similar to the tumor tissue in Crown Gall disease.
http://www.lyme-morgellons.com/morgellons.html
Based on past detailed studies of rod-shaped bacteria such as Escherichia coli and Bacillus subtilis, it has been assumed that most bacteria grow by binary fission, a dispersed mode of growth involving insertion of new cell wall material uniformly along the long axis of the cell. Growth requires breaking the cell wall at numerous places along the cylinder to allow insertion of new cell wall material, enabling uniform elongation of the cell, with the process culminated by cleavage at the mid-point of the cell to create two symmetric new cells.
The new research published today, Jan. 17, in Proceedings of the National Academy of Sciences reports on the surprising discovery that cell growth in a large group of rod-shaped bacteria occurs by insertion of new cell wall material only at a single end, or pole, of the cell rather than by the dispersed mode of growth. The cell wall of the progenitor cell remains largely intact, and all of the new cell wall material is partitioned into the new cell.
Polar growth of four bacterial species -- the plant symbiont Sinorhizobium meliloti, the plant pathogen Agrobacterium tumefaciens and the human pathogens Brucella abortus and Ochrobactrum anthropi -- was observed using time-lapse microscopy and transmission electron microscopy. The four related bacteria used in the study are all members of a large and diverse class of bacteria called the Alphaproteobacteria. The results reported suggest that polar growth is broadly distributed among many different bacterial taxa, including groups outside the Alphaproteobacteria.
There could be a number of reasons why polar growth emerged and has remained conserved and persistent in bacteria, the researchers believe. The process may act as an aid in anchoring damaged material to only the aging mother cell; it could serve as a tool for conservation of energy by constraining growth to a single region of the cell; and ensuring that newborn cells are composed of newly synthesized outer membrane proteins may help pathogens avoid detection by host immune systems.
"As a consequence of polar growth, the two bacterial cells are actually markedly different," said lead author Yves Brun, the Clyde Culbertson Professor of Biology in IU Bloomington's College of Arts and Sciences. "One cell contains all of the old cell wall and surface molecules, including those with damage. In contrast, the other cell is composed of newly synthesized, relatively pristine material."
Ensuring that some cells are composed of newly synthesized surface molecules may help bacteria vary their surface composition, and the ability to do so rapidly is thought to be advantageous for adapting to new environments. Since the defense systems of many plant and animal hosts recognize bacterial cell surfaces, rapid modification of the cell surface may allow bacteria like those used in the experiments to evade detection by the host cell's defense systems.
"These findings make it abundantly clear that the widely accepted binary fission model is not a general rule and suggest that polar growth may be broadly distributed," said IU biology professor Clay Fuqua, one of the IU co-authors. "Therefore, future work aimed at understanding the molecular mechanism underlying polar growth should provide attractive targets for the development of new antibacterial strategies."
Understanding the mechanisms of bacterial growth has enabled advances in strategies to limit the proliferation of bacteria that cause disease. Penicillin, for example, targets actively growing cells by directly inhibiting the proteins responsible for the synthesis of the cell wall and that are required for cell growth. New insights into bacterial cell growth have also been utilized to promote growth of certain bacteria used in oil spill remediation and eradicating disease-carrying mosquitoes.
http://newsinfo.iu.edu/news/page/normal/20892.html
Jan. 17, 2012
BLOOMINGTON, Ind. -- An international team of microbiologists led by Indiana University researchers has identified a new bacterial growth process -- one that occurs at a single end or pole of the cell instead of uniform, dispersed growth along the long axis of the cell -- that could have implications in the development of new antibacterial strategies.
"A different but related issue is the inability of plant tissues to respond to culture manipulations for the desired outcome (Benson 2000a). Under normal tissue culture conditions, even without transformation, many plant species fail to respond to culture manipulations (Benson 2000a). This phenomenon, referred to as “in vitro recalcitrance”, has been ascribed to several factors including cellular incompetence and necrosis but, again, the exact reason(s) for this problem remain unclear.
The three common problems described above, (1) browning/necrosis of transformed cells/tissues, (2) shoot escapes in transgenesis, and (3) in vitro recalcitrance, severely limit the number of transgenic plants that can be regenerated. Although seemingly unrelated, recent research is beginning to unravel a common factor underlying these problems, the production of ROS, which can cause growth inhibition, cell death, or alter plant metabolic pathways leading to poor regeneration of plants and the production of shoot escapes."
Biological Functions of Antioxidants in Plant Transformation - Science News - redOrbit
By Dan, Yinghui Abstract Browning and necrosis of transformed cells/ tissues, and difficulty to regenerate transgenic plants from the transformed cells/tissues (recalcitrance) are common in Agrobacterium-mediated transformation process in many plant species.
REDORBIT.COM
http://www.redorbit.com/news/science/1523653/biological_functions_of_antioxidants_in_plant_transformation/#3hfqtXO8he91bP8D.99
Autoimmunity crisis...
Caused by trying to modify Strawberries?
"Decomposition of peroxides by heating or by transition metal ion catalysis can generate both peroxyl and alkoxyl radicals (Halliwell et al. 1995). The sites and actions of antioxidants in living cells. Main antioxidant actions include scavenging ROS or free radicals, inhibiting the generation of ROS, and chelating metals, as well as their effects on cell signaling pathways and on gene expression (Halliwell et al. 1995; Soobratteea et al. 2005). However, it seems that antioxidants that interfere with the activity of OH’ do not by direct OH’ scavenging, but by scavenging or blocking the formation of its precursors (O^sup -^^sub 2^, H^sub 2^O^sub 2^, HOC1, ONOO’) and/or by binding the transition metal ions needed for OH’ formation from O^sup -^^sub 2^ and H^sub 2^O^sub 2^ (Halliwell et al. 1995). In addition, many lipid-soluble chain-breaking antioxidants can have prooxidant properties, which induce oxidative stress either through creating reactive oxygen species or inhibiting antioxidant systems, under certain circumstances in vitro, often because they can bind Fe(III) or Cu(II) ions and reduce them to Fe^sup 2+^Or Cu+ (Mukai et al. 1993).
Antioxidants act as scavengers of ROS, such as the peroxyl radical either in aqueous phase (e.g., with radicals from DNA, thiols, and proteins) or in hydrophobic phase (food lipid, membrane, lipoprotein interior) (Halliwell et al. 1995). For example, glutathione reacts rapidly with free radicals generating from an attack of OH’ on DNA, at rate constants of about 10^sup 7^-10^sup 8^ M^sup -1^ s^sup -1^ (von Sonntag 1987; Fahey 1988). Other antioxidants, e.g., scavengers of the peroxyl radical such as chain breaking antioxidants (propyl gallate and alpha-tocopherol) that are inhibitors of lipid peroxidation, could act hydrophobically in lipids, cell membranes, and interior structure of lipoproteins.
Major Problems and Antioxidants Used in Plant Tissue Culture and Transformation
Hyperhydricity. Hyperhydricity, a physiological disorder occurring in plant tissue cultures, is associated oxidative damage (Chakrabarty et al. 2006). Hyperhydricity results in a general decrease in concentrations of reduced and oxidized pyridine nucleotides, reflecting a reduction in metabolic activity (Chakrabarty et al. 2006). The activities of antioxidant enzymes, such as superoxide dismutase (SOD), catalase, ascorbate peroxidase, and glutathione reductase, were higher in hyperhydric leaves than in normal leaves, indicating that hyperhydricity was associated with oxidative stress (Chakrabarty et al. 2006). Measurements of chlorophyll fluorescence provided evidence of oxidative damage to the photosynthetic machinery in the hyperhydric leaves because the photochemical efficiency of photosystem II, the effective quantum efficiency, and photochemical quenching were all lower in the hyperhydric leaves (Chakrabarty et al. 2006). Toth et al. (2004) reported that the inclusion of glutathione in a culture medium could suppress the hyperhydricity of calli, thereby promoting regeneration of plantlets in a desiccation-tolerant plant, Ramonda myconi.
Recalcitrance. In vitro recalcitrance of plants has been associated with ROS production. Higher levels of free radical activity were found in recalcitrant genotypes of potato and grape as well as in nonembryogenic calli (rice) and reduced embryogenic capacity calli (carrot) compared to responsive genotypes and embryogenic calli, respectively, (Benson et al., 1992; Benson and Roubelakis-Angelakis 1992, 1994; Bailey et al. 1994; Bremner et al. 1997; Deighton et al. 1997). The production of the free radicals might lead to an increase in lipid peroxidation and subsequently have a negative effect on the morphogenetic capacity of plant tissue cultures (Benson 2000b).
Production of H^sub 2^O^sub 2^ coincided with the emergence of meristemoids and the formation of bud primordia in strawberry morphogenic calli (Tian et al. 2003). High O^sup -^^sub 2^ level, low H^sub 2^O^sub 2^ level, and little or no SOD activity were detected in calli possessing low organogenesis capacity. Adding N,N- diethyldithiocarbonate, an SOD inhibitor, to the regeneration medium promoted O^sup -^^sub 2^ production, inhibited H^sub 2^O^sub 2^ production and decreased the regeneration percentage, whereas the exogenous addition of H^sub 2^O^sub 2^ slightly promoted the potential for regeneration of shoot buds."
Made by DNA manipulation, or RNA interference with grapes, or sugar cane???
Monsanto is established on Maui, and the Hawaiian California Sugar corporation...HC&S grows here on Maui...And the 2 corporations are the bestest buddies every...GMO is growing in HC&S fields...And sugar cane is being modified too...
The Morgellons is a freak of nature from DNA modification or RNA interference in sugar cane...BETCHA! The strands and the strings are tough and will not die easily...And they kinda look like sugar cane fibers in a weird way to me.
Monsanto is established on Maui, and the Hawaiian California Sugar corporation...HC&S grows here on Maui...And the 2 corporations are the bestest buddies every...GMO is growing in HC&S fields...And sugar cane is being modified too...
The Morgellons is a freak of nature from DNA modification or RNA interference in sugar cane...BETCHA! The strands and the strings are tough and will not die easily...And they kinda look like sugar cane fibers in a weird way to me.
"Programmed CeU Death Associated with ROS in Plant
Mechanism of plant cell death induced by Agrobacterium. Hansen (2000) reported that the cell death triggered by Agrobacterium spp. in maize tissues had several features of apoptosis, which were DNA fragmentation, and cytological changes, and cytochrome c release. She further showed the two antiapoptotic genes from baculovirus, p35, and lap had the ability to prevent the onset of the apoptosis in maize tissues. p35 is reported to act as a direct inhibitor of caspases whereas lap may act upstream to prevent activation of caspases. Caspases control most events of apoptosis in vertebrates and in invertebrates and are responsible, either directly or indirectly, for the cleavage of cellular proteins. The proteins include nuclear proteins such as poly (ADP-ribose) polymerase, DNA- dependent protein kinase, and lamins, as well as actin (Nagata 1997). The evidence that these genes could affect the reaction of maize cells to an apoptotic stimulus strongly indicates that plant may have caspase-like proteases regulating apoptosis. A caspase- like proteolytic activity was also reported in tobacco plants undergoing an HR triggered by an infection with Tobacco mosaic virus (del Pozo and Lam 1998). Molecular identification of ROS signaling during programmed cell death (PCD) in plant. One of the first genes identified and isolated in ROS-associated plant cell death is LSDl that encodes a zinc-finger protein. This protein, together with two other zinc-finger proteins (LOLl and LOL2), could act as a molecular rheostat, sensing changes in ROS homeostasis, thereby controlling apoptosis through the regulation of apoptotic genes (Dietrich et al. 1997; Epple et al. 2003). The inactivation of the Arabidopsis Executerl gene completely abolished singlet oxygeninduced cell death (Wagner et al. 2004). The chloroplastic protein, which is encoded by Executerl, might perceive nonscavenged singlet oxygen species within the chloroplast. Other important stress signal transducers include mitogen-activated protein kinases (MAPKs) that act upstream of the oxidative burst during ozone treatment and the HR (Ren et al. 2002; Samuel and Ellis 2002). The primary ROS activated tobacco MAPK is the salicylic acidinduced protein kinase, which is required during harpindependent PCD (Samuel et al. 2005). A MAPKKK of alfalfe activates cell death induced by H^sub 2^O^sub 2^ through a specific MAPK-scaffolding action (Nakagami et al. 2004). A recent finding demonstrates the conserved Arabidopsis BCL2associated athanogene protein has been shown to be induced by H^sub 2^O^sub 2^ and capable of provoking PCD in both yeast (Saccharomyces cerevisiae) and plants (Kang et al. 2006).
Biological Functions of Antioxidants in Plant Transformation - Science News - redOrbit
By Dan, Yinghui Abstract Browning and necrosis of transformed cells/ tissues, and difficulty to regenerate transgenic plants from the transformed cells/tissues (recalcitrance) are common in Agrobacterium-mediated transformation process in many plant species.
REDORBIT.COM
http://www.redorbit.com/news/science/1523653/biological_functions_of_antioxidants_in_plant_transformation/#3hfqtXO8he91bP8D.99
Mechanism of plant cell death induced by Agrobacterium. Hansen (2000) reported that the cell death triggered by Agrobacterium spp. in maize tissues had several features of apoptosis, which were DNA fragmentation, and cytological changes, and cytochrome c release. She further showed the two antiapoptotic genes from baculovirus, p35, and lap had the ability to prevent the onset of the apoptosis in maize tissues. p35 is reported to act as a direct inhibitor of caspases whereas lap may act upstream to prevent activation of caspases. Caspases control most events of apoptosis in vertebrates and in invertebrates and are responsible, either directly or indirectly, for the cleavage of cellular proteins. The proteins include nuclear proteins such as poly (ADP-ribose) polymerase, DNA- dependent protein kinase, and lamins, as well as actin (Nagata 1997). The evidence that these genes could affect the reaction of maize cells to an apoptotic stimulus strongly indicates that plant may have caspase-like proteases regulating apoptosis. A caspase- like proteolytic activity was also reported in tobacco plants undergoing an HR triggered by an infection with Tobacco mosaic virus (del Pozo and Lam 1998). Molecular identification of ROS signaling during programmed cell death (PCD) in plant. One of the first genes identified and isolated in ROS-associated plant cell death is LSDl that encodes a zinc-finger protein. This protein, together with two other zinc-finger proteins (LOLl and LOL2), could act as a molecular rheostat, sensing changes in ROS homeostasis, thereby controlling apoptosis through the regulation of apoptotic genes (Dietrich et al. 1997; Epple et al. 2003). The inactivation of the Arabidopsis Executerl gene completely abolished singlet oxygeninduced cell death (Wagner et al. 2004). The chloroplastic protein, which is encoded by Executerl, might perceive nonscavenged singlet oxygen species within the chloroplast. Other important stress signal transducers include mitogen-activated protein kinases (MAPKs) that act upstream of the oxidative burst during ozone treatment and the HR (Ren et al. 2002; Samuel and Ellis 2002). The primary ROS activated tobacco MAPK is the salicylic acidinduced protein kinase, which is required during harpindependent PCD (Samuel et al. 2005). A MAPKKK of alfalfe activates cell death induced by H^sub 2^O^sub 2^ through a specific MAPK-scaffolding action (Nakagami et al. 2004). A recent finding demonstrates the conserved Arabidopsis BCL2associated athanogene protein has been shown to be induced by H^sub 2^O^sub 2^ and capable of provoking PCD in both yeast (Saccharomyces cerevisiae) and plants (Kang et al. 2006).
Biological Functions of Antioxidants in Plant Transformation - Science News - redOrbit
By Dan, Yinghui Abstract Browning and necrosis of transformed cells/ tissues, and difficulty to regenerate transgenic plants from the transformed cells/tissues (recalcitrance) are common in Agrobacterium-mediated transformation process in many plant species.
REDORBIT.COM
http://www.redorbit.com/news/science/1523653/biological_functions_of_antioxidants_in_plant_transformation/#3hfqtXO8he91bP8D.99
"There is quite a diversity of strange material that has unearthed itself from my body. Some of it may be what I call "throwbacks" to genetics used in original bioengineered mixtures. For more information read my "Throwback Collection of Morgellons Specimens." Other Morgellons specimens look like they are straight from a bad Sci-Fi movie or the lab of a mad scientist. The sad part of it is that I seem be to correct about the origins of Morgellons. I guess it's true that art imitates life.
Those of us with Morgellons Disease have found ourselves living in an unthinkable and ignored alternate reality. It is quite possible that everyone has Morgellons and those of us with severe reactions to it are the only people who notice. That is something for everyone to ponder. Clifford Carnicom has preliminary research that points to this conclusion. Take a look at my simple "Hairbrush Test" paper and check yourself for the tell tale signs of nanofibers.
http://www.morgellonsexposed.com/pjOddities.htm
Clifford E Carnicom Oct 15, 2011 (edited Dec 1, 2011)
Morgellons: A Thesis
Abstract:
A substantial body of research has accumulated to make the case that the underlying organism (i.e., pathogen) of the so-called "Morgellons" condition, as identified by this researcher, is using the iron from human blood for its own growth and existence. It will also be shown that the bio-chemical state of the blood is being altered in the process. The implications of this thesis are severe as this alteration affects, amongst other things, the ability and capacity of the blood to bind to oxygen. Respiration is the source of energy for the body.
This change is also anticipated to increase the number of free radicals and to increase acidity in the body. This process also requires and consumes energy from the body to take place; this energy supports the growth and proliferation of the organism. The changes in the blood are anticipated to increase its combination with respiratory inhibitors and toxins.The changes under evaluation may occur without any obvious outward symptoms. It is also anticipated that there are consequences upon metabolism and health that extend beyond the functions of the blood. This change represents essentially a systemic attack upon the body, and the difficulties of extinction of the organism are apparent. Physiological conditions that are in probable conjunction with the condition are identified. Strategies that may be beneficial in mitigating the severity of the condition are enumerated.
http://www.carnicominstitute.org/articles/bio2011-6.htm
Morgellons: a novel dermatological perspective as the multisystem infective disease borreliosis [v1; ref status: awaiting peer review,http://f1000r.es/116]
Peter Mayne1, John S English2, Edward J Kilbane3, Jennie M Burke4, Marianne J Middelveen1, Raphael B Stricker1
Abstract
Morgellons disease (MD) is a term that has been used in the last decade to describe filaments that can be found in human epidermis. It is the subject of considerable debate within the medical profession and is often labeled as delusions of parasitosis or dermatitis artefacta. This view is challenged by recent published scientific data put forward between 2011-2013 identifying the filaments found in MD as keratin and collagen based and furthermore associated with spirochetal infection. The novel model of the dermopathy put forward by those authors is further described and, in particular, presented as a dermal manifestation of the multi-system disease complex borreliosis otherwise called Lyme disease. A differential diagnosis is drawn from a dermatological perspective. The requirements for a diagnosis of delusional disorder from a psychiatric perspective are clarified and the psychological or psychiatric co-morbidity that can be found with MD cases is presented. A concurrent case incidence is also included. Management of the multisytem disease complex is discussed both in general and from a dermatological perspective. Finally replacement of the term ‘Morgellons’ by ‘borrelial dermatitis’ is proposed within the profession.
http://f1000research.com/articles/2-118#.UYJpb9wTIR4.facebookhttp://f1000research.com/articles/2-118#.UYJpb9wTIR4.facebook
This goes on and on...
Called
Yan Disease too!
Etiology of Yan (Morgellons) Disease: A New Plant-Like Infectious Organism from Brazil and Its Skin Manifestations June 19, 2012
Once on site, click on Download pdf or get it here.
This is the first article I've seen saying Morgellons is contagious.
http://www.la-press.com/journal-clinical-medicine-insights-dermatology-j69
Called
Yan Disease too!
Etiology of Yan (Morgellons) Disease: A New Plant-Like Infectious Organism from Brazil and Its Skin Manifestations June 19, 2012
Once on site, click on Download pdf or get it here.
This is the first article I've seen saying Morgellons is contagious.
Etiology of Yan (Morgellons) Disease: A New Plant-Like Infectious Organism from Brazil and Its Skin Manifestations (Published on 19 Jun 2012)
Download Full Open Access Article in PDF Format (2.60 MB)http://www.la-press.com/journal-clinical-medicine-insights-dermatology-j69
Pictured: Alice firemen suited up in their Haz-Mat gear Tuesday after a woman possibly exposed bank tellers to a contagious disease by rubbing a canister on her scabby arms. Photo by OFELIA GARCIA HUNTER
Haz-Mat team cleans up teller lane after woman threatens staff
Ofelia Garcia Hunter, Alice Echo-News Journal
A woman claiming to have Morgellons, a skin disease, drove through the Wells Fargo motor bank downtown and threatened the tellers by rubbing the bank canister on her sores and sending it through the tube.
"A customer went through the drive-thru and threatened the bank staff to give them a contagious disease," said investigator David Valadez with the police department.
Police said the lady, who was not identified and has not been arrested, had threatened the bank before.
About 2 p.m. Tuesday, the woman threatened the tellers at the motor bank with her disease called Morgellons.Police and the fire department blocked off about four lanes and the Alice Fire Department Haz-Mat team suited up and decontaminated the area, including the bank canister she apparently rubbed on her sores and the lane she used, police said.
According to the Internet, "Morgellons is an unexplained and debilitating condition that has emerged as a public health concern…Persons who suffer from this condition report a range of coetaneous symptoms including crawling, biting and stinging sensations; granules, threads or black speck-like materials on or beneath the skin; and/or skin lesions (e.g., rashes or sores) and some sufferers also report systemic manifestations such as fatigue, mental confusion, short term memory loss, joint pain and changes in vision."
This article is 7 years old...
There is a big cover-up...
There is a big cover-up...
Morgellons Disease Research & Publications
Peer Reviewed Research Publications
- Feb 12, 2015 - Exploring the association between Morgellons disease and Lyme disease: identification of Borrelia burgdorferi in Morgellons disease patients
- January 2013, PubMed - Association of Spirochetal Infection with Morgellons Disease
- January 2013, PubMed - Characterization and evolution of dermal filaments from patients with Morgellons disease
- June 12, 2012, F1000 - Morgellons Study Cited by Faculty of 1000 Study of Emerging Skin Disease Among Top 2% Published
- May 15, 2012, Clinical & Experimental Dermatology Research - Morgellons Disease: A Chemical and Light Microscopic Study
- Nov 4, 2011, PubMed - Filament formation associated with spirochetal infection: a comparative approach to Morgellons disease
Articles and Research Updates
- Morgellons: An Exercise in Medical Validation
- Discerning Between Science and Pseudoscience
- July 2013 - The CEHF funds University of New Haven's research on the Investigation of the Spirochetal Etiology of Morgellons Disease
- May 2013, F1000 - Morgellons: a Novel Dermatological Perspective as the Multisystem Infective Disease Borreliosis
- March 24, 2012 5th Annual Medical-Scientific Conference on Morgellons Disease in Austin, TX - Click here for videos with Dr. Peter Mayne and Dr. Randy Wymore discussing the Jan 2012 CDC report on Morgellons disease.
- August 22, 2012 - OSU biomedical researcher says science, not belief, will prove existence of skin disorder Morgellons
- August, 2011 - Center for the Investigation of Morgellons Disease Research Update
- July, 2009 - Center for the Investigation of Morgellons Disease Research Update
- June, 2007 - Center for the Investigation of Morgellons Disease Vision Statement
- June 19, 2007 - Position Statement on Morgellons Disease
- May 17, 2006 - Letter to Physicians
- April 17, 2006 - Morgellons Research Update: Video review request rebuttal response (and a little 'how science works' lesson)
- April 5, 2006 - Morgellons Research Update: Video review request
- December 1, 2005 - Morgellons Research Update: Progress report & Seminar brief
- October, 2005 - OSU Professor Hunts Cause of Odd Disease<
- September 9, 2005 - Morgellons Research Update: S. maltophilia & Collembola
- http://www.thecehf.org/morgellons-disease-research.html
CDC offers 1st study on Morgellons...Listen here...
http://www.krmg.com/Player/101246021/
http://www.krmg.com/Player/101246021/
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